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The SLC1A5 promoter can be directly targeted by c-Myc, causing an upregulation of SLC1A5 expression in cells and facilitating Gln uptake

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However, Aja and colleagues found no evidence for this hypothesis since in this model CPT-1 should inhibit feeding by increasing cytosolic fatty-acyl CoA levels while they actually showed the initial response of mice to a CPT-1 inhibitor was an increase in appetite [75]