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BPC-157 promotes the shift from pro-inflammatory M1 to reparative M2 macrophages, facilitating inflammation resolution and reducing scar tissue formation

Our APAP model [10] is simple because it does not include the processing of GSH in the mitochondria that has been shown to be fundamental for liver cell death in the presence of APAP [36]

After treatment, cells were washed two times with phosphate-buffered saline (DPBS

In principle, the human body can regulate the glutathione level independently

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The resulting supernatant was then transferred to a new, clean tube