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Experimental studies have demonstrated that in vitro exposure to S protein induces excessive oxidative stress in endothelial cells, which is mediated specifically by activation of NADPH oxidase isoform 2 (NOX2), but not NOX1 or NOX4 (Youn et al., 2021)

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Therapeutic potential of pro-angiogenic BPC157 is associated with VEGFR2 activation and up-regulation

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