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Thus, although GIPR in the AP is required for the weight loss effects of GIPR agonists, it is not the target of therapeutic GIPR antagonists, supporting recent findings that Gipr KO in GABAergic neurons also abolished responses to GIPR agonism but not antagonism 6
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In tendon fibroblasts, BPC-157 has been reported to increase expression of growth hormone receptor at mRNA and protein levels, potentially priming cells to respond to growth hormone via downstream Janus kinase (JAK) signaling
