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The discovery that some cancers, such as thyroid, breast, and colon cancer, exhibit a higher relative abundance of IR-A compared to normal cells provided insight into the intimate association between the insulin and IGF systems [36, 41, 42]

Early evidence already suggests that such multiplex regimens outperform single-agent therapy where metabolic heterogeneity blunts isolated responses (81, 85) (Table 3) Future work must dissect mechanism-specific dosing ratios, develop quantitative biomarkers of combinatorial efficacy, and deliver large, stratified, randomized trials to confirm both safety and clinical relevance

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[DOI] [PMC free article] [PubMed] [Google Scholar] 120.Pinget M, Goldenberg R, Niemoeller E, Muehlen-Bartmer I, Guo H, Aronson R

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