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They reflect the widespread neuroinflammation and neurodegeneration affecting brain regions beyond the obvious white matter lesions visible on MRI

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These treatments are monitored closely and adjusted based on individual response and tolerance

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In summary, the distinct receptor expression patterns and physiological roles of GLP-1, GIP, and glucagon create complementary mechanisms that, when engaged through mono-, dual-, or triple agonist formulations, address multiple pathogenic pathways in MASLD