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[4] A more potent, binding-protein-resistant analog such as IGF-1 LR3 would, by mechanism, be expected to retain this hypoglycemic potential, and because it evades the buffering system that normally tempers free IGF-1 activity, the metabolic effect could in principle be less predictable

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L.MishraN.CheX.LeeB.BatemanL.et al

These enzyme elevations were generally mild and did not exceed clinically significant thresholds