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10.1016/j.trsl.2021.10.001 123 ZhangW.MiikedaA.ZuckermanJ.JiaX.CharugundlaS.ZhouZ.et al (2021a)
This author has long recognized the need for a shift of clinical focus from glycemic status to insulin homeostasis for detecting and optimally managing adverse metabolic, proinflammatory, endothelial, developmental, and neurologic effects of hyperinsulinism (ref
Its Metabolic Daily that seems to fall short, as it contains all five species in amounts that cant possibly meet the lowest effective clinical doses
Oxidative stress, compounded by mitochondrial dysfunction and NADPH oxidase activation, damages cardiac myocytes, leading to arrhythmias and impaired ventricular function, while the formation of ONOO further disrupts cardiac contractility (117)
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