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Historically, TUDCA was associated with traditional Chinese medicine and was originally sourced from bear bile

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References: [1]Ford, Megan

62,109 These examples demonstrate that LPO and ferroptosis may be biologically irrelevant for APAP hepatotoxicity under normal conditions because endogenous defense mechanisms (such as the lipid-soluble antioxidant vitamin E and the iron chelator ferritin) effectively prevent excessive LPO