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High Met diet causes alteration of hepatic prooxidant/antioxidant status and oxidative stress parameters in rats ( Met is directly converted by MATI/MATIII into SAMe, and MATI/MATIII deficiency could lead to decreased SAMe levels and its downstream products including GSH
Apoptosis occurs in all multi-cellular organisms and is the best studied form of programmed cell death
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On the other hand, Cr FAP65AH2 and its variant, Cr FAP65AH2V12P exhibited a very high yield (3045 mg/L of E
Table 1 [4,2746])
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