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Urolithin A suppresses high glucose-induced neuronal Amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis

The airport may be easy, but a vial left in a parked car can overheat quickly

Oral administration is simple, painless, and non-invasive , making it ideal for patients hesitant about self-injection

It is essential for neutralizing free radicals, supporting liver function, and maintaining mitochondrial energy production

It has been shown to reset pathological gene expression to healthier states, which may counteract age-related dysregulation of biochemical pathways and support neuronal survival and function 11

10.3389/fphar.2020.00307 Front