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Furthermore, in signaling studies that used cell lines with recombinantly expressed human GIPR or GLP-1R, tirzepatide stimulated cyclic adenosine monophosphate (cAMP) accumulation by either receptor with potency similar to that of native GIP and approximately 13-fold weaker than that of native GLP-1 [1]

As expected, linagliptin did not increase active GLP-1 levels to control levels in the Gcg RA PDX1Cre mice as the pancreas is not a major contributor to circulating GLP-1

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