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5C) revealed that mTOR inhibition slightly impaired responsiveness to glucose, but entirely abrogated cellular ability to increase ECAR to compensate for oligomycin-induced blockade of mitochondrial ATP production

doi: 10.1111/j.1600-0625.2008.00701.x 38 YuNZhangSZuoFKangKGuanMXiangL

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