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NPH treatment markedly decreased spleen-to-body weight ratio and alleviated splenic injury induced by IMQ (Fig

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TB-500 may therefore be hypothesized to encourage angiogenesis by pairing a cytoskeleton-linked rise in endothelial motility with signaling shifts that elevate pro-angiogenic programs such as VEGF-A and Ang2/Tie2 through Notch/NF-B coupling in damaged cellular structures