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Metabolic networks in ferroptosis

In addition to de novo lipogenesis, FAO is often upregulated in tumors to meet the high energy demands of rapidly proliferating cells

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These mechanisms involve a delay in DNA damage repair, promotion of apoptosis, inhibition of angiogenesis, and regulation of the tumor microenvironment

In preclinical settings it is commonly used to explore fat-mobilisation pathways and weight-management concepts without the full growth hormone sequence

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