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High ROS levels generated via endogenous (mitochondria, endoplasmic reticulum [ER], and NOXs) and exogenous sources (radiation, drugs, foods, cigarette smoking, and pollution) induce oxidative stress that is harmful to the host 46,47

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40,41 In this review, we integrate recent advances from genetic, neuropathological, and neurobiochemical studies on ASD to further elucidate the pathogenetic mechanism at the molecular, cellular, and neural circuit levels

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