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& Hirota, J

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In relation to the primary site of infection, Ab treatment modulated the inflammatory response into peritoneal cavity of septic mice, inducing almost complete bacterial burden elimination 24 h post CLP and a significant augment in NO levels, without alterations in IL-1 and TNF- levels in relation to CLP-saline treated mice

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Recent advances in understanding NRF2 as a druggable target: development of pro-electrophilic and non-covalent NRF2 activators to overcome systemic side effects of electrophilic drugs like dimethyl fumarate