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Both insulin and IGF-1 act through their homologous receptor tyrosine kinases to coordinate metabolism and cellular responses to nutrient supply (57)

C., Kauppinen, R., Rhodes, L

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It is important to note that confirmation of ferroptosis in erythrocytes can be challenging due to the impossibility to assess characteristic alterations of organelles (especially mitochondria, absent in mature RBCs) and the complex ferroptosis signaling requiring epigenetic, transcriptional, and post-translational regulation