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The molecular mechanisms of silibinin-mediated antiproliferative effects are mainly via receptor tyrosine kinases, the androgen receptor, members of the signal transducer and activator of transcription (STAT) family, NF-kappaB (NF-B), and cell cycle regulatory and apoptotic signaling pathways in various cancer cells

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Confounding by severity and regression to the mean may be at play in the results from this study and further randomized prospective designs are needed to formally test the efficacy of these novel applications of these medications

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Adult body mass index and risk of ovarian cancer by subtype: a Mendelian randomization study