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Studies in ischemia-reperfusion injury mouse models show that ACSL4 expression is upregulated in ischemic tissues, and inhibiting ACSL4 suppresses ferroptosis by reducing lipid peroxidation, significantly mitigating tissue and organ damage [32]

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Conclusions We have developed a model of the possible competing reactions of GPX4 in reduction of lipids and production of neuromelanin, depicted in Figure 7

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