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[5] The glycerol backbone of the lipid is then able to enter the active site and is hydrolyzed

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Therapeutic implications The biology argues against indiscriminate antioxidant loading. More promising are source- and compartment-targeted strategies (e.g., NOX isoform modulation, mitochondrial quality control, ER stress tuning), buffer re-balancing (GSH/thioredoxin systems), iron management to suppress Fenton chemistry and lifestyle interventions that reshape ROS tone without blunting adaptive signalling

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