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"Instead, when cells were placed in conditions that encouraged fatty acid engagement, EPA was associated with weaker angiogenic network formation and reduced endothelial barrier integrity, matching key features of the neurovascular repair deficit seen in vivo." CTE brain reveals neurovascular and fatty acid metabolic reprogramming consistent with EPA-linked vulnerability

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2 Mechanism of action of HDACi in immunotherapy HDAC inhibitors exert complex and multifaceted immunomodulatory effects in a concentration-dependent manner by targeting key immune components in the tumor microenvironment (TME), including the extracellular matrix (ECM), macrophages, T cells, B cells, natural killer cells, and dendritic cells ( Figure 2 )