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If the clinical profile is additive rather than conflicting, this becomes an interesting scientific data point - GCGR antagonism plus GIP/GLP-1 agonism recovers the diabetes-lowering benefit the antagonist era sought while preserving the incretin-driven appetite effect

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The reduction in purine levels was associated with increased oxidative stress and endothelial dysfunction, both of which have been implicated in the pathophysiology of both ME/CFS and GWS (Furuhashi, 2020

doi: 10.3390/ijms23084404