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The performance of gonadotropins to regulate the granulosa cell ontogeny is well clarified, and the diverse fate of ovarian transplants under the stimulation of measurable gonadotropins indicates the existence of additional mechanisms of intraovarian modulation

which reported a heightened risk of thyroid cancer, particularly medullary thyroid cancer, associated with other GLP-1 RAs such as liraglutide and exenatide [133]

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