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The current review will focus on four main areas: (1) general description of oxidative stress markers in SLE, (2) alteration of redox state and complication of disease (3) role of redox mechanisms in the initiation and execution phases of apoptosis, and (4) intracellular glutathione and its checkpoints with lymphocyte apoptosis represent novel targets for pharmacological intervention in SLE

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Customized searches of proteomics data Semitryptic peptide ratios were estimated from label-free QC data searched with FragPipe (LFQ-MBR, semienzymatic)