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Overexpression of TREK-1 suppresses BDNF-mediated plasticity, and its immunoreactivity is significantly higher in TLE-D patients than in those with TLE alone, suggesting a role in inflammation-mediated neuronal damage (Li et al., 2021)

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Advances in experimental medicine and biology, 264, 7984

This bypasses the need for precursors and ensures that the "finished product" is actually available for your body to use

[DOI] [PubMed] [Google Scholar] 119.He Y, Ingudam S, Reed S, Gehring A, Strobaugh TP, Jr, Irwin P

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