You simply wouldn't use BPC-157 to study appetite suppression, just as you wouldn't use a GLP-1 agonist to study acute tendon repair
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The triple-receptor action (GLP-1, GIP, glucagon) is the reason it outperforms semaglutide and tirzepatide
In animal studies, administration of BPC 157 following the damage caused by these drugs has shown that damage such as ventricular tachycardia, bradycardia, T wave elevation, QTc prolongation and asystole can be completely resolved in mice
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