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The same angiogenesis that helps a damaged tendon can in theory help a tumor get a blood supply, which is why oncologists pay attention to this mechanism even though no study has shown BPC-157 causes cancer

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The N-methyl-d-aspartate (NMDA) receptor-mediated excitatory procedure, which provides the intracellular Ca2+ increase necessary for nNOS activation, is another mechanism by which UCB-induced neuronal damage occurs

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