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Semaglutide primarily activates GLP-1 receptors, while tirzepatide activates both GLP-1 and GIP receptors simultaneously
This suggests a causal role of CD36 in steatosis supported by abnormally increased CD36 levels in NAFLD patients: a small study reported increased CD36 mRNA levels in obese subjects with high amounts of intracellular fat (66.0 6.8%, n = 5), compared to subjects with low liver fat content (6.4 2.7%, n = 5) [24]
According to the study, GLP-1 medicines reduced body fat in obese mice much more than lean body mass, and fat and liver mass fell faster than skeletal muscle
XV, 73-118 (1957)
Frequently asked questions Is it normal for tirzepatide with B12 to be bright pink
GLP-1 drugs and thrombotic risk
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