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doi: 10.1089/thy.2015.0592 98 LiDLiangGCalderoneRBellantiJA

A final alternative to alleviate FcRn-controlled pathological mechanisms could be to alter FcRn in itself, as may be intended with a gene therapy being developed in a collaboration between Beam and Apellis for a one-time dose of gene-editing molecules targeting FcRn [436, 437]

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falciparum glutathione redox system comprises the electron donor NADPH, a highly active PfGR located in the cytosol and the apicoplast, and reduced/oxidized glutathione (GSH/GSSG) [21], [22], [23]