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Fourteen patients were lost to follow-up and eventually the data of 83 patients were analyzed

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Moreover, overexpression of GPX3 further exerted anti-autophagy by inhibiting AMPK/m-TOR and up-regulated nuclear factor erythroid 2-related factor 2 (Nrf2)/glutathione peroxidase 4 (GPX4, mitochondrial GPX4 and cytoplasmic GPX4) to antagonize autophagy-related ferroptosis

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By managing inflammation and speeding up tissue repair, KLOW can be a game-changer for your performance