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During myocardial reperfusion following ischemia, the production of oxygen free radicals increases dramatically, disrupting oxidative balance and antioxidant defense systems, leading to cellular damage and abnormal immune responses ( Mitochondrial dysfunction is a hallmark of myocardial I/R injury, with impaired mitochondrial quality control serving as a critical factor in arrhythmia development (Tu et al., 2013)
10.1073/pnas.1112063108 4 AlexandrovP
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