The reciprocal substitution significantly decreased GLP-1Rs rate of internalization but not to the same extent to that of the wild-type GIPR
TBI pathogenesis involves a myriad of post-injury responses and secondary damage to brain tissues, culminating in cellular damage and loss [4]
This degeneration allows vertebrae to move closer together, potentially pinching nerve roots
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Kaplan-Meier analysis (Figure 2) showed early separation of cumulative incidence curves, which progressively widened over time