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Histological changes are similar to those observed in the Reye's syndrome, with early production of microvesicular steatosis followed by development of centri-lobular necrosis [23]

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Treatment remains the same

Raising growth hormone levels causes the body to produce more IGF-1, a related hormone that supports growth and tissue repair

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One of the reasons for this was the poor activity of microbial GSTs with CDNB - 1-chloro-2,4-dinitrobenzene as a model substrate for GST activity, which led to the conclusion that these enzymes are rare in unicellular organisms [81], [77]