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Palmitoyl tripeptide-1 primarily works through matrikine receptor-mediated TGF-beta signaling without the copper-dependent effects

Compliance note: this catalog text is informational and does not provide medical advice, administration instructions, or treatment claims.

Thereby, it is important to indicate that with consistently less nerve damage in all damaged areas in all BPC 157-cuprizone rats, the most important evidence is the consistent beneficial effect of BPC 157 also in those areas that were most affected, and thereby markedly improved gross presentation and functions in BPC 157 rats

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