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Failure to complete apoptosis following neonatal hypoxia-ischemiamanifests as "continuum" phenotype of cell death and occurs with multiple manifestations of mitochondrial dysfunction in rodent forebrain

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Pharmacokinetic analysis showed that plasma BPC-157 concentrations returned to baseline within 24 h, consistent with its known rapid clearance and short half-life [52]

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Their lengths tend to diminish with repeated cell replication and aging [2]