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Thus, data in support of each hypothesis was derived from different animal models, with mitochondrial oxidative stress being critical for APAP hepatotoxicity in mice on a normal diet, without appreciable LPO generation

Experimental infection of cell lines has also shown that the activities of antioxidant enzymes may decrease over time 18,19 and that chemical stimulation from reactive oxygen species may even induce an upregulation of antioxidant enzymes in order to counteract oxidative stress 17

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Cunningham and Porat-Shliom, 2021)

In: Al-Suhaimi EA (eds) Emerging Concepts in Endocrine Structure and Functions