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This conjugate is recognized by specific transporters and is carried to the intercellular medium where it undergoes action of -glutamyl transpeptidase (GT) which removes the glutamic acid residue [32]

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Consistently, in vitro studies have shown that silencing TXNIP reduces autophagic vesicle accumulation and the expression of LC3-II and p62 in renal tubular cells exposed to high glucose, thereby enhancing autophagic flux and resolving autophagic dysfunction (150)

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325 At the cellular level this overexcitation stems from a slow synchronous depolarisation of neurones, known as paroxysmal depolarisation shift (PDS) within the epileptic foci