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The histopathological features of SJS/TEN are extensive necrosis of epidermal keratinocytes, mainly mediated by drug-specific cytotoxic T cells [18]

Paradoxically, while NOX2 inhibition exacerbates inflammation by disrupting redox-metabolic homeostasis, targeted activation restores metabolic balance and alleviates pathology, indicating strategic ROS modulationnot suppressionas a promising therapeutic paradigm for psoriasis

In the oxidant recovery assay both mitochondrial sensors behaved like the cytosolic sensorfollowing diamide washout they recovered rapidly to the respective steady states with similar kinetics (Figure 2B)

Benefits: improved brain metabolism, protection from oxidative stress, and enhanced recovery from age-related or stress-induced cognitive decline

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