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Complementing this primary mechanism, emerging evidence indicates that SFN modulates iron metabolism through upregulation of ferritin heavy chain (FTH1), potentially altering intracellular iron homeostasis to promote iron-mediated lipid peroxidation via Fenton chemistry (69, 70)
(Eur J Endocrinol, 1998
Complications of Carnitine Deficiency Syndrome include organ damage to the liver, heart, brain, and various skeletal muscles of the body, including heart muscle weakness (cardiomyopathy) Brain injury may present severe brain dysfunction (encephalopathy), irreversible learning problems, and mental retardation Another complication includes problems with the metabolism of the body, especially relating to fat and energy metabolism
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