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Even though oral treatments of the methyl donor SAM ameliorated ASD-associated deficits in valproic acid (VPA)-exposed mice by reversing epigenetic changes, there has been limited success thus far in the development of preventative agents similar to SAM for the clinical management of ASD in humans

MeSH terms searched included SGLT2 or sodium glucose transport proteins, GLP-1 or glucagon-like peptides, and T2DM