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Alteration of several metabolic enzymes and pathways that affect KG abundance and KGDD activity have been reported in cancer, such as mutations of IDH1 and IDH2, changes in glutaminolysis that deplete KG, or alterations in fumarate hydratase (FH) and succinate dehydrogenase (SDH) that inhibit KGDD activity (Losman et al., 2020)

Metaphases analysis To assess single-chromosome damage, HCT116 Wild type (WT) and ADH5 cells were plated in P60 dishes, allowed to adhere and then treated with mitomycin C (MMC, Santa Cruz, #sc-3514) 0.5 g mL 1 during 24 h or with FA 150 mol L 1 during 48 h

Chen Y, Wu S, Tian Y