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Many treatments are available for cerebral edema and intracranial hypertension

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4af), indicating that lysosomes are responsible for the degradation of SERCA2a during TMAO-induced cardiomyocyte hypertrophy

JMF4073 eliminates WT-Bcr-Abl-transformed myeloid cells in vitro and in vivo Since the balanced supply of dNTPs is critical for overcoming DNA damage and replication stress for cell survival, we then tested whether JMF4073 treatment could selectively suppress the growth of Bcr-Abl-transformed but not untransformed myeloid cells