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MUC2, a core barrier component and key regulatory target, is suppressed under dual inhibition by ischemia-activated HIF-1 and TNF--mediated NF-B signaling, which decreases MUC2 promoter activity and hinders its transcription/expression in intestinal epithelial cells (Liu et al., 2023)

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A shortage in riboflavin can lead to a notable increase in the loss of iron via the gastrointestinal tract and a reduction in the release of iron from its body storages [102]