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This metabolic wiring generates oxidizable PUFA-phospholipid species that can subsequently undergo enzymatic peroxidation (for example, via lipoxygenases) or non-enzymatic oxidation driven by iron-dependent reactive oxygen species, thereby establishing a lipid network that amplifies ferroptotic vulnerability

Not only in euthyroid rats but also in rats with experimentally induced hypothyroidism, iodine treatment increased serum level of lipid peroxidation products and decreased NIS gene expression [183]

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In a study published by Blundell et al

Friedman, J.M

These losses occurred irrespective of stress model or duration