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The authors show that the drastic growth arrest of the sir1 mutant is partially rescued when glutathione deficiency is introduced via the cad2 mutation, which decreases glutathione levels to 30% to 40% of wild-type levels (Cobbett et al., 1998), or pharmacologically through treatment with the GCL inhibitor buthionine sulfoximine
What does this mean
So, if we do not have enough glutathione, we do not have the building blocks for our cells to work properly, including to detox the body, or create crucial chemicals in the body, such as the stomach acid we need to adjust our food or the melatonin we need to sleep at night properly
2017 Oct 1;186:66-79
Key limitation: With only 3 patients per group, even a single unusually responsive or non-responsive patient could substantially skew the results
