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Tan WC, Lo C, Jong A, Xing L, Fitzgerald MJ, Vollmer WM, Buist SA, Sin DD

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Intellectual disorder (ID), ASD, ADHD, schizophrenia, and bipolar disorder (BD) display a neurodevelopmental continuum that can be explained by gene nucleotide substitutions and copy number variants, e.g., nonsense mutations or splice variants

Along with mitochondria, synapses are considered end effectors of many molecular mechanisms related to ASD, and the convergence of different neurodevelopmental pathological mechanisms on synapses can at least partially explain the behavioral similarities in different individuals on the spectrum

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The extensive role of GLP-1Rs and their agonists in other organs and tissues are shown in the overview in Fig