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PAGln induces endothelial cell activation through increased reactive oxygen species leading to endothelial dysfunction To elucidate vascular endothelial dysfunction, we cultured HCAECs and exposed them to varying concentrations and durations of PAGln (Fig

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PLoS ONE 10:e0126312

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However, the remaining portion is converted by cytochrome P450 enzymes into N-acetyl-p-benzoquinone imine (NAPQI), a toxic metabolite

YTHDF1 knockdown reduced SLC7A5 protein levels without affecting mRNA expression