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Clinical investigations into the cardiovascular benefits of UA remain limited, with the majority of current evidence originating from in vitro and animal models
Pombo und Kollegen zeigten, dass die maximale GH-Antwort auf GHRP-6 endogenes hypothalamisches GHRH voraussetzt (Pombo et al., 1998)
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The pro-inflammatory triggers apparently drove up TNF- and IL-6 release, whereas GHK-Cu apparently pulled both cytokines down
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