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severe pancreatitis depends on whether the inflammatory response resolves or amplifies, leading to multi-organ failure.[ref] More to the pancreatitis story than trypsin: While the activation of trypsin in the pancreas is a driving factor in initiating the damage in pancreatitis, it may not be the only causative factor

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When glucose or ATP levels decline, the AMPK becomes activated and promotes catabolic processes to restore energy homeostasis and attenuate cellular senescence ( Figure 1 )